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6–0 fine middle cerebral artery occlusion (mcao) suture  (Doccol Corporation)

 
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    Doccol Corporation 6–0 fine middle cerebral artery occlusion (mcao) suture
    Loss of CLEC14A <t>exacerbated</t> <t>cerebral</t> injury with VEGFR2 activation in <t>MCAO</t> stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD
    6–0 Fine Middle Cerebral Artery Occlusion (Mcao) Suture, supplied by Doccol Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/6–0 fine middle cerebral artery occlusion (mcao) suture/product/Doccol Corporation
    Average 90 stars, based on 1 article reviews
    6–0 fine middle cerebral artery occlusion (mcao) suture - by Bioz Stars, 2026-03
    90/100 stars

    Images

    1) Product Images from "CLEC14A deficiency exacerbates neuronal loss by increasing blood-brain barrier permeability and inflammation"

    Article Title: CLEC14A deficiency exacerbates neuronal loss by increasing blood-brain barrier permeability and inflammation

    Journal: Journal of Neuroinflammation

    doi: 10.1186/s12974-020-1727-6

    Loss of CLEC14A exacerbated cerebral injury with VEGFR2 activation in MCAO stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD
    Figure Legend Snippet: Loss of CLEC14A exacerbated cerebral injury with VEGFR2 activation in MCAO stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD

    Techniques Used: Activation Assay, Comparison, Injection, Spectrophotometry, Staining, Expressing



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    6–0 fine middle cerebral artery occlusion (mcao) suture  (Doccol Corporation)
    90
    Doccol Corporation 6–0 fine middle cerebral artery occlusion (mcao) suture
    Loss of CLEC14A <t>exacerbated</t> <t>cerebral</t> injury with VEGFR2 activation in <t>MCAO</t> stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD
    6–0 Fine Middle Cerebral Artery Occlusion (Mcao) Suture, supplied by Doccol Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/6–0 fine middle cerebral artery occlusion (mcao) suture/product/Doccol Corporation
    Average 90 stars, based on 1 article reviews
    6–0 fine middle cerebral artery occlusion (mcao) suture - by Bioz Stars, 2026-03
    90/100 stars
    		  Buy from Supplier

    Image Search Results


    Loss of CLEC14A exacerbated cerebral injury with VEGFR2 activation in MCAO stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD

    Journal: Journal of Neuroinflammation

    Article Title: CLEC14A deficiency exacerbates neuronal loss by increasing blood-brain barrier permeability and inflammation

    doi: 10.1186/s12974-020-1727-6

    Figure Lengend Snippet: Loss of CLEC14A exacerbated cerebral injury with VEGFR2 activation in MCAO stroke model. a , c Images of whole brains and comparison of Evans blue dye (EB) leakage in brains (P8 and adult) after EB injection (intraperitoneal route) in WT and CLEC14A KO mice ( n = 6, each group). b , d The quantitative analysis of Evans blue (EB) dye extravasation using spectrophotometer, 24 h after intraperitoneal injection of EB dye. e TTC staining of brain slices in the sham, WT, and CLEC14A KO groups 24-h post ischemia-reperfusion. Severe infarction was present in the CLEC14A KO group. Scale bar = 1 mm. f Percentage of infarct volume in the WT and CLEC14A KO groups 24 h after ischemia-reperfusion. g Neurological scores of WT and CLEC14A KO mice were evaluated. h VEGFR2, pVEGFR2 protein expression in ipsilateral brain lysate after ischemia-reperfusion injury. i , j Quantification data of relative pVEGFR2 and VEGFR2 protein expression. n = 6 per group; * P < 0.05, ** P < 0.01, *** P < 0.001. The results are mean values and the error bars represent the mean ± SD

    Article Snippet: A 6–0 fine middle cerebral artery occlusion (MCAO) suture (Doccol Corporation, Sharon, MA) was inserted into the lumen of the right external carotid artery stump.

    Techniques: Activation Assay, Comparison, Injection, Spectrophotometry, Staining, Expressing